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• Direct myocardial cell injury may occur by disruption of mitochondria and induction of intrafibrillar collagen dysplasia (Melchert and Welder 1995; Sullivan et al 1998).
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• We report a case of an acute ST-segment elevation myocardial infarction in a affected person with traditional cardiac risk factors utilizing supraphysiologic doses of supplemental, intramuscular testosterone.
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Moreover, a part of endothelial dysfunction has been proposed, which may contribute to abnormal vessel reactivity. However, testosterone in physiologic doses could even be useful in patients with angina , have larger ischemic thresholds and improved high quality of life . Short-term intracoronary administration of testosterone induces coronary artery dilation and increases coronary blood move in males with established coronary artery disease . Therefore, an interplay of endogenous components of the patient’s danger profile in addition to the testosterone dosing could also be issues of whether or not opposed events happen.

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